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NMDA receptors solicit flux-independent intra-cellular Ca2+ signs through metabotropic glutamate receptors as well as flux-dependent nitric oxide supplement discharge within brain microvascular endothelial tissue.
NMDA receptors solicit flux-independent intra-cellular Ca2+ signs through metabotropic glutamate receptors as well as flux-dependent nitric oxide supplement discharge within brain microvascular endothelial tissue.